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Pituitary function was assessed in a woman with primary hyperaldosteronism. Basal plasma levels of oestradiol-17 beta, luteinizing hormone, follicle stimulating hormone, and prolactin were normal. Serum aldosterone was at the low end of the normal range and decreased at 30 min after a 100 mEq. sodium chloride infusion (P less than 0.001) and at 1 and 2 hr after a 1 mg. dexamethasone infusion (P less than 0.05 and P less than 0.01). After oral sodium loading, plasma aldosterone increased from the normal range to the hypertensive range. Plasma aldosterone decreased after 2 weeks of bupropion or placebo medication and returned to the normal range after 1 week of treatment with chlorthalidone. These data demonstrate that: (1) hyponatremia can stimulate pituitary secretion of prolactin; (2) the dexamethasone suppression test can be used to suppress excess production of pituitary ACTH. in hyperaldosteronism; and (3) bupropion may be effective in the treatment of this condition.Molecular cloning and characterization of an alcohol dehydrogenase gene from Streptomyces davydovii MT-1.
Overexpression of an inducible Streptomyces albus alcohol dehydrogenase (ADH) gene in S. davydovii MT-1 has recently been shown to improve ethanol yield in S. albus [Ennos et al. (2002) Appl. Microbiol. Biotechnol. 59, 576-583]. A S. davydovii strain was cultured with 1% ethanol in the medium for 20 h at 30 °C. The strain produces four isoforms of ADH with molecular masses of 109 kDa, 83 kDa, 59 kDa and 24 kDa. By using PCR cloning and heterologous expression, an ADH gene encoding the 59 kDa isoform in the strain was amplified. Its nucleotide sequence was determined. Its deduced amino acid sequence and expressed protein in the recombinant E. coli were